Cannabis tötet Krebszellen. Was in den U.S.-amerikanischen Mainstreammedien bereits in der breiten Masse dokumentiert und zelebriert wird, stellt die Sensation (bzw. der Skandal, je nach Perspektive des historischen Erzählers) dar, dass Cannabis von gewissen Krebsarten heilen kann, wovon gemäss amtlichen Regierungsdokumenten gewisse Mitglieder der U.S.-Regierungen bereits seit dem Jahr 1974 wussten, aber bewusst verschwiegen.
Den krebsheilenden Umstand verifizierende wissenschaftliche Studien sowohl bei Einsatz von natürlichem wie auch von synthetischem Medizinalcannabis sind untenstehend aufgeführt.
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Update (Mai 2014):
http://www.google.com/patents/US20130059018
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SINCE 1974 THE GOVERNMENT HAVE KNOWN THAT THE CHEMICALS FOUND IN THE CANNABIS PLANT CAN BE USED TO TREAT CANCER
PLEASE LOOK BELOW FOR PEER REVIEWED STUDIES FROM AROUND THE GLOBE WHICH CONFIRM THESE FINDINGS
These same chemicals can have a unique effect on each vital organ, homeostatically and holistically aiding them in their roles. This includes the kidneys and the nephrological system.
Many scientific studies have reported that various cannabinoids (both natural and synthetic) exert a wide range of growth-inhibiting effects on cancer cells, including:
- Triggering cell death, through a mechanism called apoptosis.
- Stopping cells from dividing.
- Preventing new blood vessels from growing into tumours –a process termed angiogenesis.
- Reducing the chances of cancer cells to metastasize through the body, by stopping cells from moving or invading neighbouring tissue.
- Speeding up the cell’s internal ‘waste disposal machine’ –a process known as autophagy – which can lead to cell death.
http://cannabis-med.org/index.php?tpl=faq&red=faqli st&id=274&lng=en
PEER REVIEWED SCIENTIFIC SRUDIES REGARDING CANNABIS AND CANCER:
Cannabinoid receptor systems: therapeutic targets for tumour intervention. UK
http://www.ncbi.nlm.nih.gov/ pubmed/14640910
High concentrations of cannabinoids activate apoptosis in human U373MG glioma cells. UK
http://www.ncbi.nlm.nih.gov/ pubmed/18615640
Cannabinoid receptor agonists are mitochondrial inhibitors: a unified hypothesis of how cannabinoids modulate mitochondrial function and induce cell death. UK
http://www.ncbi.nlm.nih.gov/ pubmed/17931597
The cannabinoid delta(9)-tetrahydrocannabinol inhibits RAS-MAPK and PI3K-AKT survival signalling and induces BAD-mediated apoptosis in colorectal cancer cells. UK
http://www.ncbi.nlm.nih.gov/ pubmed/17583570
Cannabis-induced cytotoxicity in leukemic cell lines: the role of the cannabinoid receptors and the MAPK pathway. UK
http://www.ncbi.nlm.nih.gov/ pubmed/15454482
Targeting cannabinoid receptors to treat leukemia: role of cross-talk between extrinsic and intrinsic pathways in Delta9-tetrahydrocannabinol (THC)-induced apoptosis of Jurkat cells. USA
http://www.ncbi.nlm.nih.gov/ pubmed/15978942
The expression level of CB1 and CB2 receptors determines their efficacy at inducing apoptosis in astrocytomas. USA
http://www.ncbi.nlm.nih.gov/ pubmed/20090845
Cannabidiol-induced apoptosis in human leukemia cells: A novel role of cannabidiol in the regulation of p22phox and Nox4 expression. USA
http://www.ncbi.nlm.nih.gov/ pubmed/16754784
Cannabidiol enhances the inhibitory effects of delta9-tetrahydrocannabinol on human glioblastoma cell proliferation and survival. USA
http://www.ncbi.nlm.nih.gov/ pubmed/20053780
Cannabinoids and the immune system. USA
http://www.ncbi.nlm.nih.gov/ pubmed/11854771
Delta(9)-tetrahydrocannabinol-induced apoptosis in the thymus and spleen as a mechanism of immunosuppression in vitro and in vivo. USA
http://www.ncbi.nlm.nih.gov/ pubmed/12130702
Delta9-tetrahydrocannabinol-induced apoptosis in Jurkat leukemia T cells is regulated by translocation of Bad to mitochondria. USA
http://www.ncbi.nlm.nih.gov/ pubmed/16908594
Cannabinoid-induced apoptosis in immune cells as a pathway to immunosuppression. USA
http://www.ncbi.nlm.nih.gov/ pubmed/19457575
Synthetic cannabinoid receptor agonists inhibit tumor growth and metastasis of breast cancer. USA
http://www.ncbi.nlm.nih.gov/ pubmed/19887554
Cannabinoids for cancer treatment: progress and promise. USA
http://www.ncbi.nlm.nih.gov/ pubmed/18199524
Targeting CB2 cannabinoid receptors as a novel therapy to treat malignant lymphoblastic disease. USA
http://www.ncbi.nlm.nih.gov/ pubmed/12091357
Cannabinoid receptor as a novel target for the treatment of prostate cancer. USA
http://www.ncbi.nlm.nih.gov/ pubmed/15753356
In vivo effects of cannabinoids on macromolecular biosynthesis in Lewis lung carcinomas. USA
http://www.ncbi.nlm.nih.gov/ pubmed/616322
Cannabidiol as a novel inhibitor of Id-1 gene expression in aggressive breast cancer cells. USA
http://www.ncbi.nlm.nih.gov/ pubmed/18025276
Cannabinoids as novel anti-inflammatory drugs. USA
http://www.ncbi.nlm.nih.gov/ pubmed/20191092
De novo-synthesized ceramide is involved in cannabinoid-induced apoptosis. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/11903061
Amphiregulin is a factor for resistance of glioma cells to cannabinoid-induced apoptosis. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/19229996
Cannabinoids and cell fate. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/12182964
Delta9-tetrahydrocannabinol induces apoptosis in human prostate PC-3 cells via a receptor-independent mechanism. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/10570948
Delta9-tetrahydrocannabinol induces apoptosis in C6 glioma cells. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/9771884
Cannabinoids inhibit glioma cell invasion by down-regulating matrix metalloproteinase-2 expression. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/18339876
Cannabinoids protect astrocytes from ceramide-induced apoptosis through the phosphatidylinositol 3-kinase/protein kinase B pathway. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/12133838
Hypothesis: cannabinoid therapy for the treatment of gliomas? SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/15275820
Involvement of cannabinoids in cellular proliferation. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/15638794
Effects on cell viability. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16596790
Cannabinoids induce apoptosis of pancreatic tumor cells via endoplasmic reticulum stress-related genes. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16818650
Cannabinoids and gliomas. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/17952650
Delta9-tetrahydrocannabinol inhibits cell cycle progression in human breast cancer cells through Cdc2 regulation. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16818634
Opposite changes in cannabinoid CB1 and CB2 receptor expression in human gliomas. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/20307616
Inhibition of skin tumor growth and angiogenesis in vivo by activation of cannabinoid receptors. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/12511587
Inhibition of human tumour prostate PC-3 cell growth by cannabinoids R(+)-Methanandamide and JWH-015: involvement of CB2. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/19690545
Control of the cell survival/death decision by cannabinoids. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/11269508
The stress-regulated protein p8 mediates cannabinoid-induced apoptosis of tumor cells. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16616335
The CB2 cannabinoid receptor signals apoptosis via ceramide-dependent activation of the mitochondrial intrinsic pathway. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16624285
p38 MAPK is involved in CB2 receptor-induced apoptosis of human leukaemia cells. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16139274
Cannabinoids and ceramide: two lipids acting hand-by-hand. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/15958274
Endocannabinoids: a new family of lipid mediators involved in the regulation of neural cell development. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/16787257
Anti-tumoral action of cannabinoids: involvement of sustained ceramide accumulation and extracellular signal-regulated kinase activation. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/10700234
Cannabinoid receptors as novel targets for the treatment of melanoma. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/17065222
JunD is involved in the antiproliferative effect of Delta9-tetrahydrocannabinol on human breast cancer cells. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/18454173
Cannabinoid action induces autophagy-mediated cell death through stimulation of ER stress in human glioma cells. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/19425170
Cannabinoids induce glioma stem-like cell differentiation and inhibit gliomagenesis. SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/17202146
Down-regulation of tissue inhibitor of metalloproteinases-1 in gliomas: a new marker of cannabinoid antitumoral activity? SPAIN
http://www.ncbi.nlm.nih.gov/ pubmed/17675107
Endocannabinoids and fatty acid amides in cancer, inflammation and related disorders. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/11106791
Estrogenic induction of cannabinoid CB1 receptor in human colon cancer cell lines. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/18938775
Antitumor effects of cannabidiol, a nonpsychoactive cannabinoid, on human glioma cell lines. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/14617682
The endocannabinoid anandamide neither impairs in vitro T-cell function nor induces regulatory T-cell generation. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19189659
Cannabinoid derivatives induce cell death in pancreatic MIA PaCa-2 cells via a receptor-independent mechanism. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/16500647
The CB1/CB2 receptor agonist WIN-55,212-2 reduces viability of human Kaposi's sarcoma cells in vitro. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19539619
Antitumor activity of plant cannabinoids with emphasis on the effect of cannabidiol on human breast carcinoma. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/16728591
Cannabinoids in intestinal inflammation and cancer. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19442536
The endocannabinoid system as a target for the development of new drugs for cancer therapy. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/12723496
Apoptosis induced in HepG2 cells by the synthetic cannabinoid WIN: involvement of the transcription factor PPARgamma. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19059457
Endocannabinoids as emerging suppressors of angiogenesis and tumor invasion (review). ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/17342320
The non-psychoactive cannabidiol triggers caspase activation and oxidative stress in human glioma cells. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/16909207
Cannabinoids as potential new therapy for the treatment of gliomas. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/18088200
The endogenous cannabinoid anandamide inhibits human breast cancer cell proliferation. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/9653194
Anandamide induces apoptosis in human cells via vanilloid receptors. Evidence for a protective role of cannabinoid receptors. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/10913156
Cannabinoid receptor activation induces apoptosis through tumor necrosis factor alpha-mediated ceramide de novo synthesis in colon cancer cells. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19047095
Plant-derived cannabinoids modulate the activity of transient receptor potential channels of ankyrin type-1 and melastatin type-8. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/18354058
A metabolically stable analogue of anandamide, Met-F-AEA, inhibits human thyroid carcinoma cell lines by activation of apoptosis. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/19189054
Endocannabinoids in the immune system and cancer. ITALY
http://www.ncbi.nlm.nih.gov/ pubmed/12052046
R(+)-methanandamide-induced apoptosis of human cervical carcinoma cells involves a cyclooxygenase-2-dependent pathway. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/19015962
Antitumorigenic effects of cannabinoids beyond apoptosis. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/19889794
Up-regulation of cyclooxygenase-2 expression is involved in R(+)-methanandamide-induced apoptotic death of human neuroglioma cells. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/15361550
Cannabinoid receptors in human astroglial tumors. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/16893424
Cannabinoids induce cancer cell proliferation via tumor necrosis factor alpha-converting enzyme (TACE/ADAM17)-mediated transactivation of the epidermal growth factor receptor. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/15026328
Cannabidiol inhibits cancer cell invasion via upregulation of tissue inhibitor of matrix metalloproteinases-1. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/19914218
Pharmacokinetics and pharmacodynamics of cannabinoids. GERMANY
http://www.ncbi.nlm.nih.gov/ pubmed/12648025
Delta 9-tetrahydrocannabinol inhibits cell cycle progression by downregulation of E2F1 in human glioblastoma multiforme cells. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/17934890
Cannabinoids and cancer. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/16250836
Cannabinoids in health and disease. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/18286801
A cannabinoid quinone inhibits angiogenesis by targeting vascular endothelial cells. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/16571653
Gamma-irradiation enhances apoptosis induced by cannabidiol, a non-psychotropic cannabinoid, in cultured HL-60 myeloblastic leukemia cells. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/14692532
An endogenous cannabinoid (2-AG) is neuroprotective after brain injury. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/11586361
HU-331, a novel cannabinoid-based anticancer topoisomerase II inhibitor. ISRAEL
http://www.ncbi.nlm.nih.gov/ pubmed/17237277
Predominant CB2 receptor expression in endothelial cells of glioblastoma in humans. SWITZERLAND
http://www.ncbi.nlm.nih.gov/ pubmed/19480992
Cannabinoid receptor 1 is a potential drug target for treatment of translocation-positive rhabdomyosarcoma. SWITZERLAND
http://www.ncbi.nlm.nih.gov/ pubmed/19509271
Cannabinoid receptor ligands as potential anticancer agents--high hopes for new therapies? SWITZERLAND
http://www.ncbi.nlm.nih.gov/ pubmed/19589225
Arachidonylethanolamide induces apoptosis of human glioma cells through vanilloid receptor-1. SWITZERLAND
http://www.ncbi.nlm.nih.gov/ pubmed/15453094
Expression of cannabinoid receptors type 1 and type 2 in non-Hodgkin lymphoma: growth inhibition by receptor activation. SWEDEN
http://www.ncbi.nlm.nih.gov/ pubmed/18546271
Cannabinoid receptor-mediated apoptosis induced by R(+)-methanandamide and Win55,212-2 is associated with ceramide accumulation and p38 activation in mantle cell lymphoma. SWEDEN
http://www.ncbi.nlm.nih.gov/ pubmed/16936228
Cannabinoid receptor ligands mediate growth inhibition and cell death in mantle cell lymphoma. SWEDEN
http://www.ncbi.nlm.nih.gov/ pubmed/16337199
Potentiation of cannabinoid-induced cytotoxicity in mantle cell lymphoma through modulation of ceramide metabolism. SWEDEN
http://www.ncbi.nlm.nih.gov/ pubmed/19609004
The endocannabinoid system of the skin in health and disease: novel perspectives and therapeutic opportunities. HUNGARY
http://www.ncbi.nlm.nih.gov/ pubmed/19608284
Pharmacological synergism between cannabinoids and paclitaxel in gastric cancer cell lines. JAPAN
http://www.ncbi.nlm.nih.gov/ pubmed/19394652
Different views on the association between cannabinoids and cancer. SLOVAKIA
http://www.ncbi.nlm.nih.gov/ pubmed/16835997
Effect of a synthetic cannabinoid agonist on the proliferation and invasion of gastric cancer cells. KOREA
http://www.ncbi.nlm.nih.gov/ pubmed/20336665
Cannabinoid 2 receptor induction by IL-12 and its potential as a therapeutic target for the treatment of anaplastic thyroid carcinoma. SAUDI ARABIA
http://www.ncbi.nlm.nih.gov/ pubmed/18197164
Cannabinoids in the treatment of cancer. NEW ZEALAND
http://www.ncbi.nlm.nih.gov/ pubmed/19442435
Cannabinoids down-regulate PI3K/Akt and Erk signalling pathways and activate proapoptotic function of Bad protein. POLAND
http://www.ncbi.nlm.nih.gov/ pubmed/15451022
Antineoplastic and apoptotic effects of cannabinoids. N-acylethanolamines: protectors or killers? UKRAINE
http://www.ncbi.nlm.nih.gov/ pubmed/18438336
A comparative study on cannabidiol-induced apoptosis in murine thymocytes and EL-4 thymoma cells. TAIWAN
http://www.ncbi.nlm.nih.gov/ pubmed/18387516
The dual effects of delta(9)-tetrahydrocannabinol on cholangiocarcinoma cells: anti-invasion activity at low concentration and apoptosis induction at high concentration. THAILAND
http://www.ncbi.nlm.nih.gov/ pubmed/19916793 —
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